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Yokochi7edicionpdf13

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Yokochi7edicionpdf13

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youtube.com/watch?v=m9yI22HOMIwVitamin D metabolism in humans.
The vitamin D sterols (vitamin D3 and vitamin D2), which are fat-soluble micronutrients, are generated endogenously via the action of ultraviolet light on 7-dehydrocholesterol in the skin and via the action of ultraviolet light on 7-dehydrocholesterol in the liver. When ingested, vitamin D2 or, in the case of some synthetic vitamin D analogues, vitamin D3, are absorbed, transported to the liver, where they undergo hydroxylation to the active forms, 1,25-dihydroxyvitamin D (1,25(OH)2D) and 24,25-dihydroxyvitamin D (24,25(OH)2D) (Vitamin D3-A), respectively. The physiologically active metabolite, 1,25(OH)2D, which is bound to the vitamin D receptor (VDR), plays a key role in calcium homeostasis. The physiologically active metabolite, 1,25(OH)2D, which is bound to the vitamin D receptor (VDR), plays a key role in calcium homeostasis. 24,25(OH)2D is metabolized in the kidney by the 24-hydroxylase enzyme and converted to the inactive 1,24,25(OH)3D that is eventually secreted into the urine. More recently, it has been demonstrated that 24,25(OH)2D also exerts potent effects via binding to the VDR and then recruiting nuclear factor-kappaB (NF-kappaB) to the vitamin D responsive element of the c-myc promoter. The effect of active vitamin D3 on the proliferation of human breast cancer MCF-7 cells appears to be mediated via an inhibition of estrogen synthesis. The effects of the therapeutic administration of active vitamin D3 to bone mineral density and bone density are now being investigated.Attention: This news story is somewhat outdated

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